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Commentary: Aortopulmonary Collaterals in Hypoplastic Left heart Syndrome: Chicken or the Egg?

  • Timothy S. Lancaster
    Affiliations
    Section of Pediatric Cardiovascular Surgery, Department of Cardiac Surgery, C.S. Mott Children's Hospital/University of Michigan, Ann Arbor, MI
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  • Vikram Sood
    Correspondence
    Address reprint requests to Vikram Sood, MD, C.S. Mott Children's Hospital, University of Michigan; 1540 E. Hospital Drive, Fl. 11, Rm. 743, Ann Arbor, MI 48109, Tel.: 734-936-4978, Fax: 734-232-8595.
    Affiliations
    Section of Pediatric Cardiovascular Surgery, Department of Cardiac Surgery, C.S. Mott Children's Hospital/University of Michigan, Ann Arbor, MI
    Search for articles by this author
Published:September 20, 2022DOI:https://doi.org/10.1053/j.semtcvs.2022.09.004
      The mechanism for development and pathophysiology of aortopulmonary collaterals in HLHS remains unclear.
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      Linked Article

      • Impact of Anatomical Sub-types and Shunt Types on Aortopulmonary Collaterals in Hypoplastic Left Heart Syndrome
        Seminars in Thoracic and Cardiovascular Surgery
        • Preview
          This study aims to clarify the relation of development of aortopulmonary collateral arteries (APCs) with anatomical sub-types and the shunt types at Norwood procedure in patients with hypoplastic left heart syndrome (HLHS). A total of 140 patients with HLHS who completed 3 staged palliation between 2003 and 2019 were included. Incidence of APCs and corresponding interventions were examined using angiogram by cardiac catheterization, with respect to the anatomical sub-types and shunt types. Totally, APCs were observed in 87 (62%) of the patients; pre-stage II in 32 (23%), pre-stage III in 64 (46%), and after stage III in 40 (29%).
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